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Although its precise etiology remains elusive, converging evidence implicates maladaptive plasticity within pain-processing networks, characterized by impaired descending inhibitory pathways, enhanced excitatory neurotransmission, and sustained activation of glial cells [8]

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HEPATOCYTE CELL DEATH MECHANISMS AFTER APAP TOXICITY The mode of cell death after APAP-induced liver injury was controversial initially, since a number of mechanistic steps in the process, such as mitochondrial Bax translocation, release of cytochrome c64,79,88, and nuclear DNA fragmentation32, were similar to that seen during apoptosis (Fig